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virus resistance vs viral resistance

Both 'virus resistance' and 'viral resistance' are correct phrases, but they are used in different contexts. 'Virus resistance' refers to the ability of an organism to resist a virus, while 'viral resistance' is more commonly used in the context of drug resistance against viruses.

Last updated: March 30, 2024 • 437 views

virus resistance

This phrase is correct and commonly used in the context of an organism's ability to resist a virus.

This phrase is used to describe the ability of an organism to resist a virus or viral infection.

Examples:

  • The plant showed strong virus resistance against the new strain.
  • Genetic modifications can enhance virus resistance in crops.
  • Doing so could increase the risk of the virus developing resistance.
  • This way, you can be sure your medicine is fully effective and you reduce the risk of the virus developing resistance to the treatment.
  • This may mean that the HIV virus will develop resistance to ISENTRESS and become harder to treat.
  • Threats such as pandemic influenza, malaria and tuberculosis are a health risk because of virus mutations, increasing resistance to antibiotics and weak health systems.
  • At time of failure with CXCR4-using virus, the resistance pattern to other antiretrovirals appears similar to that of the CCR5-tropic population at baseline, based on available data.
  • The virus is significant in its extraordinary resistance to environmental factors.
  • Background resistance to other classes of antiretrovirals have been shown to be similar in previously undetected CXCR4-tropic virus of the minor viral population, as that found in CCR5-tropic virus.
  • Resistance to entecavir (when a virus becomes insensitive to the antiviral) has been seen in lamivudine-refractory patients.
  • Resistance In vitro selection of darunavir-resistant virus from wild type HIV-1 was lengthy (> 3 years).
  • HBV strains expressing the adefovir-associated resistance mutations rtA181V and rtN236T showed a susceptibility to tenofovir ranging from 2.9- to 10-fold that of wild-type virus.
  • Viral resistance to abacavir develops relatively slowly in vitro, requiring multiple mutations for a clinically relevant increase in EC50 over wild-type virus.
  • The phenotypic resistance test performed required a minimum of 1000 copies/ ml HIV RNA in order to be able to amplify the virus.
  • In cell based assays, HBV strains expressing the rtV173L, rtL180M, and rtM204I/ V mutations associated with resistance to lamivudine and telbivudine showed a susceptibility to tenofovir ranging from 0.7- to 3.4-fold that of wild-type virus.
  • HBV strains expressing the rtL180M, rtT184G, rtS202G/ I, rtM204V and rtM250V mutations associated with resistance to entecavir showed a susceptibility to tenofovir ranging from 0.6- to 6.9-fold that of wild- type virus.
  • As the virus has the capability to develop resistance to antiretroviral agents, it is increasingly important to monitor the development of resistant strains. Insufficient laboratory capacity can be an obstacle to producing relevant susceptibility data.
  • 10 vitro studies indicate that zidovudine-resistant virus isolates can become zidovudine sensitive when they simultaneously acquire resistance to lamivudine.
  • Scientists fear that massive use of this will lead to considerable environmental pollution of the natural microflora and a significant build-up of resistance in the virus strains.
  • Phenotypic resistance: in patients with CCR5-tropic virus at time of treatment failure with CELSENTRI, 22 out of 58 patients had virus with reduced sensitivity to maraviroc.
  • Its extreme infectiousness and high degree of resistance, which vary according to the strain of the virus in question, call for the taking of draconian measures in infected areas.
  • Their genetic make-up was modified via the implantation of the Medusa gene, which means that they become fluorescent when in contact with blue light, and the Rhesus macaque gene which grants them resistance to the AIDS virus.

Alternatives:

  • resistance to viruses
  • immune to viruses
  • virus immunity
  • virus resilience
  • virus tolerance

viral resistance

This phrase is correct and commonly used in the context of drug resistance against viruses.

This phrase is used to describe the resistance of viruses to drugs or treatments.

Examples:

  • The development of viral resistance to antiviral drugs is a major concern.
  • The virus showed high levels of viral resistance to the medication.
  • Prior to randomization, OBT were selected by the investigator based on the patient's prior treatment history, as well as baseline genotypic and phenotypic viral resistance testing.
  • The choice of REYATAZ in treatment experienced patients should be based on individual viral resistance testing and the patient's treatment history (see section 5.1).
  • The choice of amprenavir should be based on individual viral resistance testing and treatment history of patients (see section 5.1).
  • In protease inhibitor (PI) experienced patients the choice of nelfinavir should be based on individual viral resistance testing and treatment history.
  • All patients received an optimised background (OB) regimen consisting of 3 to 5 antiretroviral agents selected on the basis of the patient's prior treatment history, as well as baseline genotypic and phenotypic viral resistance measurements.
  • We've discovered with AIDS we need cocktails to work well so that the viral resistance - we need several anti-virals.
  • In protease inhibitor (PI) experienced patients the choice of Telzir should be based on individual viral resistance testing and treatment history (see section 5.1).
  • The choice of Viread to treat antiretroviral experienced patients with HIV-1 infection should be based on individual viral resistance testing and/ or treatment history of patients.
  • The choice of Kaletra to treat protease inhibitor experienced HIV-1 infected patients should be based on individual viral resistance testing and treatment history of patients (see sections 4.4 and 5.1).
  • Viral resistance to abacavir develops relatively slowly in vitro, requiring multiple mutations for a clinically relevant increase in EC50 over wild-type virus.
  • VIRAMUNE should not be used as the sole active antiretroviral, as monotherapy with any antiretroviral has shown to result in viral resistance.
  • Taking all doses at the appropriate times greatly increase the effectiveness of your medicinal product combination regimen and reduces the development of viral resistance.
  • If you stop taking VIRAMUNE Taking all doses at the appropriate times greatly increase the effectiveness of your medicinal product combination regimen and reduce the development of viral resistance.
  • Viral resistance Following in vitro selection of ganciclovir-resistant HCMV isolates, cross-resistance between ganciclovir and cidofovir was seen with ganciclovir-selected mutations in the HCMV DNA polymerase gene but not with mutations in the UL97 gene.
  • Yields, water requirements, viral resistance.
  • The OBT was selected on the basis of the subject's prior treatment history and baseline genotypic and phenotypic viral resistance measurements.

Alternatives:

  • antiviral resistance
  • drug resistance in viruses
  • resistance of viruses to treatment
  • virus insensitivity to drugs
  • virus tolerance to medication

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