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i have a fast vs i am in fasting

These two phrases are not comparable as they have different meanings. 'I have a fast' is not a complete sentence and doesn't make sense on its own. 'I am in fasting' is also not a common or correct way to express the idea of fasting. It would be better to say 'I am fasting' to convey the meaning of abstaining from food or certain activities for a period of time.

Last updated: March 29, 2024 • 1869 views

i have a fast

This phrase is not correct in English. It is not a complete sentence and doesn't convey a clear meaning.

This phrase is not commonly used in English. It would be better to provide more context or use a different expression to convey the intended meaning.
  • no today I have a fast you also have a fast?
  • Look, I just have a fast question, Father.
  • Look, I just have a fast question,.
  • Man, have I got a fast food joint for you to wear that to.
  • I might have to make a fast getaway.
  • I haven't worked in a fast-food restaurant in years.
  • I have a really fast metabolism, and it works quicklier than other people's.
  • I have a federal fast pass, so I get to jump the line.
  • I have a sure and fast belief in Christ my Lord and the salvation of his blood.
  • I have met a more 'fast.
  • I have a tendency to move fast.
  • Retail is a fast-paced and exciting world.
  • Schwimmer's dad's pulling a fast one.
  • Applications for capacity-building projects will follow a fast-track award procedure.
  • A fast response is of critical importance.
  • It is an important point, because asylum seekers are entitled to a fast decision and a fast answer on their future status.
  • They did a fast rope insertion 60 miles away.
  • It was a fast decision which we must take as positive.
  • He thinks he can pull a fast one.
  • The core strength of a fast bowler.

i am in fasting

This phrase is not a common or correct way to express the idea of fasting in English.

Instead of 'i am in fasting,' it would be more appropriate to say 'I am fasting' to convey the idea of abstaining from food or certain activities for a period of time.
  • There was also a small increase in fasting blood glucose and in total cholesterol in duloxetine-treated patients while those laboratory tests showed a slight decrease in the routine care group.
  • The tenofovir disoproxil fumarate arm also had significantly smaller mean increases in fasting triglycerides and total cholesterol than the comparator arm.
  • The enhanced increase in the insulin/glucagon ratio during hyperglycaemia due to increased incretin hormone levels results in a decrease in fasting and postprandial hepatic glucose production, leading to reduced glycaemia.
  • Clinical trials data The glucose lowering effects observed with rosiglitazone are gradual in onset with near maximal reductions in fasting plasma glucose (FPG) evident following approximately 8 weeks of therapy.
  • In clinical trials, the glucose lowering effects observed with rosiglitazone are gradual in onset with near maximal reductions in fasting plasma glucose (FPG) evident following approximately 8 weeks of therapy.
  • Reduction in fasting plasma glucose (FPG) was observed at 3 weeks, the first time point at which FPG was measured.
  • Mean increases in fasting lipid values (total cholesterol, LDL cholesterol, and triglycerides) were greater in patients without evidence of lipid dysregulation at baseline.
  • This is more marked in fasting state than in fed state.
  • In the 12 week acute phase of three clinical trials of duloxetine in patients with diabetic neuropathic pain, small but statistically significant increases in fasting blood glucose were observed in duloxetine- treated patients.
  • No clinically relevant differences were seen in the pharmacokinetics of repaglinide, when repaglinide was administered 0, 15 or 30 minutes before a meal or in fasting state.
  • The magnitude of changes in fasting total cholesterol, LDL cholesterol, triglycerides, and
  • Changes in fasting glucose from borderline at baseline (>= 5.56- < 7 mmol/ l) to high (>= 7 mmol/ l) were very common.
  • Changes in fasting triglycerides from borderline at baseline (>= 1.69 mmol/ l- < 2.26 mmol/ l) to high (>= 2.26 mmol/ l) were very common.
  • The magnitude of changes in fasting total cholesterol, LDL cholesterol, triglycerides, and prolactin (see sections 4.4 and 4.8) were greater in adolescents than in adults.
  • Changes in fasting triglycerides from borderline at baseline (>= 1.69 mmol/ l - < 2.26 mmol/ l) to high (>= 2.26 mmol/ l) were very common.
  • Food appears to increase pharmacokinetic variability relative to fasting.
  • You can drink water during the fasting period.
  • Prolonged fasting before administration should be avoided.
  • In the 12 week acute phase of three clinical trials of duloxetine in patients with diabetic neuropathic pain, small but statistically significant increases in fasting blood glucose were observed in duloxetine- treated patients HbA1c was stable in both duloxetine-treated and placebo-treated patients.
  • Co-administration with a high-fat meal did not affect the AUC of atazanavir relative to fasting conditions and the Cmax was within 11% of fasting values.

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